Synaptic Plasticity (2)
Professor Graham Collingridge explains that synaptic plasticity is the way most information is stored in the central nervous system.
Synaptic plasticity is the way in which we believe that most information is stored in the central nervous system. So, we have millions upon millions of neurons, which make on average 10,000 connections with each other, giving us about 10 to the [power of] 15 synapses. The way in which synapses communicate information isnâ€™t constant with time but can be changed â€“ it can be modified by plasticity.
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Professor Graham Collingridge briefly describes how the NMDA receptor facilitates Hebbian learning (a mechanism of synaptic plasticity).
Professor Graham Collingridge describes the process of long-term potentiation (LTP) - the process by which synapses increase their efficiency.
Professor Graham Collingridge describes the process of long-term depression (LTD), a way of decreasing the efficiency of synaptic transmission.
Professor Graham Collingridge describes the glutamate receptor, AMPA, the workhorse receptor for communicating information.
Professor Tom O'Dell comments that phosphorylation plays a crucial role in synaptic plasticity.
Professor Graham Collingridge describes the roles played by NMDA and AMPA receptors in long-term potentiation (LTP).
Professor Tom O'Dell defines depotentiation - the erasure of long-term potentiation (LTP) at the synapse.
Professor Jeff Lichtman examines the concept of synaptic plasticity, a term that refers to the way the brain changes.
Professor Earl Miller explains that that the term 'plasticity' is used by neuroscientists to refer to the fact that the brain changes as a result of experience.
Professor Tom O'Dell discusses synaptic plasticity - the strengthening and weakening of synaptic connections between neurons.